With syncope, if it gets far enough to full knock me out, there’s residual muscle weakness for a few hours after. It’s short (45-60 secs) and when it resolves, the muscles don’t feel residual weakness. A psychological condition marked by severe decreases or increases of movement. However, they are not the definite answer to the problem. My cataplexy, otoh, is only triggered by two different factors and is a very specific muscle group that reacts. A sudden loss of muscle tone and strength, usually caused by an extreme emotional stimulus. They underline the significance of the support they lend to the diagnosis. The findings of the objective tests done on our patients are in agreement with those described by other authors. Regarding TMLS, the average latency < 5 mn is a parameter met by all cases, although one did not have SOREMP's. Of the HLA types, DQ1 was present in all our patients, unlike DR2 which was not found in two patients. In this paper we review our series of cases consisting of fourteen patients who fulfil the clinical diagnostic criteria required in the ICSD-1990. However, the exact diagnostic significance of some aspects of these tests and their parameters is still under discussion. Objective tests, such as typing for HLA, DR2DQ1 (DRw15DQw6, WHO90) and above all TMLS (average latency < 5 mn and two or more onsets of sleep in the phases REM and SOREMP's) is of great help. Sometimes by living with or experiencing a disorder, you can be the source of a wealth of information. Diagnosis is based on the clinical findings, although this may be difficult especially with respect to confirming the cataplexy. Cataplexy: ' A sudden loss of muscle control with retention of clear consciousness that follows a strong emotional stimulus (as elation, surprise or anger) and is a characteristic symptom of narcolepsy.' Merriam-Webster Medical Dictionary. Because narcolepsy is an under-recognised disease, it is important that general practitioners and other primary health-care workers identify abnormal daytime sleepiness early.The narcolepsy-cataplexy syndrome is a disorder of unknown aetiology, characterized by excessive daytime sleepiness associated with cataplexy and other REM sleep phenomena. It has been observed in historical figures such as Socrates, St. ![]() It can also be caused by schizophrenia treatment, anesthetic ketamine, or hypnosis. Treatment is with stimulant drugs to suppress daytime sleepiness, antidepressants for cataplexy, and gamma hydroxybutyrate for both symptoms. Catalepsy is a nervous condition of muscular rigidity and fixity of posture, often caused by certain nervous disorders or conditions such as Parkinson's disease, epilepsy, cocaine withdrawal, or catatonia. The cause of neural loss could be autoimmune since most patients have the HLA DQB1*0602 allele that predisposes individuals to the disorder. ![]() Pathophysiological studies have shown that the disease is caused by the early loss of neurons in the hypothalamus that produce hypocretin, a wakefulness-associated neurotransmitter present in cerebrospinal fluid. Catalepsy: Passive induction of postures held against gravity Waxy flexibility: Slight and even resistance to repositioning by the examiner Stupor: No psychomotor activity, no reactivity to the. The onset of narcolepsy with cataplexy is usually during teenage and young adulthood and persists throughout the lifetime. medicalknowledgeonline, what is Cataplexy ,what is Catalepsy ,what are the Difference Between Cataplexy and Catalepsy medicalknowledgeonlineTO. Sleep monitoring during night and day shows rapid sleep onset and abnormal, shortened rapid-eye-movement sleep latencies. It is characterised by severe, irresistible daytime sleepiness and sudden loss of muscle tone (cataplexy), and can be associated with sleep-onset or sleep-offset paralysis and hallucinations, frequent movement and awakening during sleep, and weight gain. Narcolepsy with cataplexy is a disabling sleep disorder affecting 0.02% of adults worldwide.
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